Carmen J. Williams

This study investigates how neonatal exposure to diethylstilbestrol disrupts uterine epithelial polarity and partial EMT state in mice, leading to long-term functional impairments and cancer risk.

The paper shows that calcium signals at fertilization alter metabolic substrates, affecting epigenetic marks H3K27ac and H3K18la, which impact embryo development and offspring health.

This study identifies a mechanism by which estrogen receptor alpha suppresses PRICKLE1, leading to REST loss and promoting uterine fibroid development.

This study identifies DUXBL as a key factor promoting the silencing of the 2C transcriptional program during exit from totipotency.

The paper investigates how developmental exposure to diethylstilbestrol (DES) disrupts uterine differentiation and leads to adenocarcinoma through Wnt/β-catenin and PI3K/AKT signaling.

This study tests whether superovulation affects calcium oscillations in fertilized eggs and finds no differences in calcium signaling patterns between superovulated and naturally cycling mice.