Harrison J. Stratton

The paper identifies a novel subset of IL-1R1+ nociceptors in mice using multimodal transcriptomics and calcium imaging, revealing their role in inflammatory pain.

This study investigates how anti-CV2/CRMP5 autoantibodies cause pain in paraneoplastic neurological syndromes and identifies potential treatments.

The paper identifies a unique NaV1.7 domain that can be targeted to produce analgesia by disrupting its interaction with CRMP2.

The paper shows that female human nociceptors are sensitized by prolactin, indicating a female-selective pain mechanism.

This study shows that SARS-CoV-2 Spike protein blocks VEGF-A/NRP-1 signaling, reducing pain and potentially contributing to asymptomatic transmission.

This study shows that preventing CRMP2 SUMOylation reduces mechanical allodynia in female neuropathic pain models but not in males or other pain types.