Débora Foguel

This study shows that neutrophil extracellular traps (NETs) degrade α-synuclein amyloid fibrils in vitro and in vivo, improving lung function by clearing these fibrils from lung tissue.

The paper shows that alpha-synuclein oligomers activate NFAT, leading to neuronal toxicity and synaptic loss, which can be reversed by NFAT inhibitors.

This study shows that oligomeric α-synuclein reduces keratinocyte proliferation and induces inflammation in a reconstructed human epidermis model.