Hongjun Wang

This study shows that AAT-overexpressing MSCs reverse T1D in NOD mice by enhancing Treg function and inducing CD8+ T cell exhaustion.

This study identifies defective mesenchymal Bmpr1a-mediated BMP signaling as a cause of congenital pulmonary cysts by disrupting lung branching morphogenesis and airway structure.

The paper identifies TRIP13 as a mediator of AT2 cell dysfunction in IPF through integrated transcriptomic and epigenomic analysis and shows TRIP13 inhibition prevents mesenchymal gene signature acquisition.