Author

Simote Totauhelotu Foliaki

National Institutes of HealthORCID 0000-0001-7450-6926Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Organism:mouse· 1 Assay:protein structure· 1

Frequent Co-Authors

Cathryn L. HaighiD· 2 shared Andreu Matamoros‐AnglesiD· 1 shared Audrey E. ThurmiD· 1 shared Behnam MohammadiiD· 1 shared Benjamin H. SchwarziD· 1 shared Berta PuigiD· 1 shared Bibiana BielekovaiD· 1 shared Carole CrozetiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Cathryn L. HaighORCID

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2 papers

Recent·Most cited

NeuroscienceJournal of Clinical InvestigationPeer-reviewedAug 4, 2026

Proteasome mutations associated with CANDLE syndrome cause altered neuronal development by dysregulating polyamine synthesis

Clayton W. Winkler, Benjamin H. Schwarz, Katie Williams…·Karin E. Peterson

This study shows that proteasome mutations in CANDLE syndrome disrupt neuronal development by increasing polyamine levels through ODC dysregulation, and inhibiting ODC can reverse these effects.

DOI 10.1172/jci199140

humanspatial transcriptomics

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NeuroscienceActa NeuropathologicaPeer-reviewedJul 9, 2024

Cleavage site-directed antibodies reveal the prion protein in humans is shed by ADAM10 at Y226 and associates with misfolded protein deposits in neurodegenerative diseases

Feizhi Song, Valerija Kovač, Behnam Mohammadi…·Hermann C. Altmeppen

This study identifies ADAM10-mediated shedding of human PrP at Y226 and shows sPrP associates with misfolded protein aggregates in neurodegenerative diseases.

9 citations

humanmouse

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