Author

Stephen W. G. Tait

Cancer Research UK Scotland InstituteORCID 0000-0001-7697-132XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Alejandro Huerta UribeiD· 1 shared Alexander CollingsiD· 1 shared Alicia C. BorgeaudiD· 1 shared Amandine Stooss· 1 shared Anand ManoharaniD· 1 shared Calvin KleiniD· 1 shared Christina HalseyiD· 1 shared Daniela Ross‐KaschitzaiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Alejandro Huerta UribeORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintDec 17, 2025

Thiol Scarcity in Cerebrospinal Fluid Renders Leptomeningeal Acute Lymphoblastic Leukaemia Therapeutically Vulnerable to Ferroptosis

Nikolai Gajic, Rhona Christie, Shirley Lam…·Christina Halsey

This paper identifies a metabolic vulnerability in leptomeningeal ALL cells due to thiol scarcity in CSF, making them susceptible to ferroptosis through disruption of selenocysteine biosynthesis and GPX4 activity.

DOI 10.64898/2025.12.15.693383

humanbulk RNA-seq

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Cell BiologyNature CommunicationsPeer-reviewedOct 24, 2025

Large transient assemblies of Apaf1 constitute the apoptosome in cells

Alicia C. Borgeaud, Iva Ganeva, Calvin Klein…·Wanda Kukulski

The paper shows that Apaf1 forms transient foci during apoptosis, which correspond to the apoptosome and regulate apoptosis progression.

15 citations

imagingexperimental assay

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