Hongbin Li

This study shows that blocking the Src-PSD-95 interaction with a peptide rescues NMDAR hypofunction and improves behavioral deficits in schizophrenia models.

This paper describes a CRISPR/Cas9-based method to create a mouse model with a tandem duplication of Irak1 and Mecp2, which exhibits MDS-like neurological and immune phenotypes.

This study shows that alternative splicing of the GluN1 subunit affects glycine priming of NMDA receptors, influencing their internalization and synaptic dynamics.