Author

Eric M. Green

Maze (United States)ORCID 0000-0001-7874-9482Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:human· 3 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Georgiana MilleriD· 3 shared Aaron D. GitleriD· 2 shared Caiwei GuoiD· 2 shared Dennis W. DicksoniD· 2 shared Leonard PetrucelliiD· 2 shared Ryan D. MorrieiD· 2 shared Sarat C. VatsavayaiiD· 2 shared Shila Mekhoubad· 2 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Georgiana MillerORCID

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3 papers

Recent·Most cited

TDP-43 dysfunction induces cryptic circular RNAs in ALS/FTD

Dario Dattilo, Flaminia Pellegrini, Simone Barattucci, Anna‐Leigh Brown, Jose Norberto S. Vargas, Ariana P. Gatt, Ryan D. Morrie, Georgiana Miller, Iris Bachmutsky, Zachary T. McEachin, Mingee Chung, Matthew J. Keuss, Eugeni M. Ryadnov, Matteo Zanovello, Puja R. Mehta, , Pietro Fratta

This study identifies TDP-43 dysfunction as a cause of cryptic circular RNA formation in ALS/FTD, showing their association with disease-relevant protein loss and potential as biomarkers.

bioRxiv · Sep 20, 2026 · PreprintNeuroscience

Cryptic splicing in synaptic and membrane excitability genes links TDP-43 loss to neuronal dysfunction

Caiwei Guo, Kuchuan Chen, Sarat C. Vatsavayai, Tetsuya Akiyama, Chang Liu, Yi Zeng, Odilia Sianto, Edith Yang, Juliane Bombosch, Rasheen Powell, Shannon Zhen, Shila Mekhoubad, Ryan D. Morrie, Georgiana Miller, Dragana Ilic, , Aaron D. Gitler

This study identifies TDP-43-dependent cryptic splicing events in synaptic and membrane excitability genes and shows they drive neuronal dysfunction in ALS and FTD.

Science Translational Medicine · Jun 3, 2026Peer-reviewedNeuroscience

TDP-43 represses cryptic exon inclusion in FTD/ALS gene UNC13A

X. Rosa Ma, Mercedes Prudencio, Yuka Mitsuhashi Koike, Sarat C. Vatsavayai, Garam Kım, Fred Harbinski, Adam Briner, Caitlin M. Rodriguez, Caiwei Guo, Tetsuya Akiyama, Hermann Broder Schmidt, Beryl B. Cummings, David W. Wyatt, Katherine Kurylo, Georgiana Miller, , Aaron D. Gitler

This study shows that TDP-43 represses a cryptic exon in UNC13A, and genetic variants associated with FTD/ALS increase cryptic exon inclusion when TDP-43 is dysfunctional.

Nature · Feb 23, 2022Peer-reviewedNeuroscience