Author

Catherine Vaurs‐Barrière

Centre National de la Recherche ScientifiqueORCID 0000-0001-8654-4292Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:statistical model· 1

Frequent Co-Authors

Franck CourtiD· 2 shared Philippe ArnaúdiD· 2 shared Aaron Bogutz· 1 shared Alice Murigneux· 1 shared Amanda Ha· 1 shared Anne FogliiD· 1 shared Anne-Charlotte FromagetiD· 1 shared Bertille MontibusiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersMolecular Biology

Franck CourtORCID

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2 papers

Recent·Most cited

The elongation of Mest transcript into MestXL sustains, but does not initiate, the maternal allele bias of its convergent gene Copg2 during neurogenesis

Sophie Perillous, Anne-Charlotte Fromaget, Céline Gonthier‐Guéret, Ornella Clerici, Marion Espenel, Alice Murigneux, Sophie Phan, Léa Feit, Catherine Vaurs‐Barrière, Floric Slimani, Céline Lemmers, Davide Normanno, Amanda Ha, Nolan Ashworth, Aaron Bogutz, , Philippe Arnaúd

The paper investigates the regulation of maternal allele bias in Copg2 during neurogenesis, showing it involves a two-step mechanism beyond MestXL-mediated interference.

Nature Communications · Sep 23, 2026Peer-reviewedMolecular Biology

Most of transcriptional alterations in glioma result from DNA-methylation independent mechanisms

Franck Court, Elisa Le Boiteux, Anne Fogli, Mélanie Müller-Barthélémy, Catherine Vaurs‐Barrière, Emmanuel Chautard, Bruno Pereira, J. Biau, Jean‐Louis Kémény, Toufic Khalil, Lucie Karayan, Pierre Verrelle, Philippe Arnaúd

The paper investigates the role of DNA methylation and H3K27me3 in transcriptional alterations in glioma, finding that H3K27me3 defects are more prevalent than DNA methylation defects in gene deregulation.

bioRxiv · Feb 1, 2019Molecular Biology