Agnès H. Michel

This study investigates the genetic basis of cellular learning in budding yeast, showing it arises from a dynamic, redundant network with variable gene contributions and broad functional integration.

This study shows that respiration-induced gene expression and stress response pathways are critical for maintaining cytoplasmic properties and survival in yeast quiescence.

The paper investigates how the pro-apoptotic lipid t-2-hex disrupts mitochondrial protein import and proteostasis in yeast, identifying key mechanisms and targets.

This study uses SATAY to identify antifungal resistance mechanisms and key determinants of ATI-2307 and Chitosan sensitivity in yeast.

The paper shows SUMO factors regulate DPC repair pathways in yeast, promoting repair without Tdp1 but inhibiting it when Wss1 is absent.

This study identifies Ddi1 as a protease involved in DNA-protein crosslink repair in yeast, showing its role in evicting crosslinked proteins from DNA.