Author

Bryan E. Abuchery

Universidade de São PauloORCID 0000-0001-9637-4637Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Anuj Kumar ShuklaiD· 1 shared Bruno Alves SantarossaiD· 1 shared Carolina Moura Costa Catta‐PretaiD· 1 shared Craig T. LapsleyiD· 1 shared Eduardo M. R. ReisiD· 1 shared Fernanda Merchiori GerolamoiD· 1 shared Henning Jacob JesseniD· 1 shared Marcelo S. da SilvaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMicrobiology

Anuj Kumar ShuklaORCID

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1 paper

Recent·Most cited

MicrobiologybioRxivPreprintMay 29, 2026

Reduced levels of inositol hexakisphosphate kinase (IP6K) impair life-cycle transitions and the intracellular development of Trypanosoma cruzi within human cardiomyocytes

Bryan E. Abuchery, Thaíse Lara Teixeira, Vitor Luiz da Silva…·Marcelo S. da Silva

This study shows that reducing IP6K levels in T. cruzi impairs key life-cycle transitions and intracellular development in human cardiomyocytes, suggesting IP6K as a potential drug target for Chagas disease.

DOI 10.64898/2026.01.21.700787

genetic perturbation / CRISPRhost-pathogen & immune response

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