Author

Thaise Lara Teixeira

Institute of Chemistry, University of Sao Paulo (USP);ORCID 0000-0002-5541-0387Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Marcelo S. da SilvaiD· 2 shared Vitor Luiz da SilvaiD· 2 shared Anuj Kumar ShuklaiD· 1 shared Bruno Alves SantarossaiD· 1 shared Bryan E. AbucheryiD· 1 shared Carolina Moura Costa Catta‐PretaiD· 1 shared Craig T. LapsleyiD· 1 shared Eduardo M. R. ReisiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersMolecular Biology

Marcelo S. da SilvaORCID

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2 papers

Recent·Most cited

A comparative analysis of BrdU and EdU genotoxicity shows that trypanosomatids exhibit unique responses to the presence of these thymidine analogs in their DNA

Marcos Antonio Fernandes de Oliveira, Vitor Luiz da Silva, Thaise Lara Teixeira, Fernanda Merchiori Gerolamo, Tarcillo J. N. Gaziri, Jennifer Ann Black, Fernanda de Jesus Silva, Hernan Chaimovich, Iolanda Midea Cuccovia, Marcelo S. da Silva

This study compares the genotoxic effects of BrdU and EdU in trypanosomatids, finding EdU more genotoxic and revealing species-specific responses to DNA damage.

bioRxiv · Nov 3, 2026 · PreprintMolecular Biology

Reduced levels of inositol hexakisphosphate kinase (IP6K) impair life-cycle transitions and the intracellular development of Trypanosoma cruzi within human cardiomyocytes

Bryan E. Abuchery, Thaíse Lara Teixeira, Vitor Luiz da Silva, Rauni Borges Marques, Fernanda Merchiori Gerolamo, Anuj Kumar Shukla, Carolina Moura Costa Catta‐Preta, Bruno Alves Santarossa, Craig T. Lapsley, Yete Gambarini Ferri, María Cristina M. Motta, Samuel Cota Teixeira, Miguel Ángel Chiurillo, Noelia Marina Lander, Simone Guedes Calderano, , Marcelo S. da Silva

This study shows that reducing IP6K levels in T. cruzi impairs key life-cycle transitions and intracellular development in human cardiomyocytes, suggesting IP6K as a potential drug target for Chagas disease.

bioRxiv · May 29, 2026 · PreprintMicrobiology