Author

Peer Lukat

Helmholtz Centre for Infection ResearchORCID 0000-0001-9897-3430Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:CRISPR screen· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Wulf BlankenfeldtiD· 2 shared Agnes BartelsiD· 1 shared Angela MiguriD· 1 shared Anuja KibeiD· 1 shared Chase L. BeiseliD· 1 shared Emely JockmanniD· 1 shared Göksu Oral· 1 shared Gunhild LayeriD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBiochemistry

Wulf BlankenfeldtORCID

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2 papers

Recent·Most cited

BiochemistryChemBioChemPeer-reviewedJul 26, 2026

S-Adenosyl-D-methionine as a Non-Physiological Substrate for a Wide Range of SAM-Dependent Enzymes

Philipp Germer, Lukas Gericke, Lars-Hendrik Koeppl…·Jennifer N. Andexer

This study investigates the ability of SAM-dependent enzymes to accept D-SAM instead of L-SAM, finding that several enzymes can utilize D-SAM and highlighting implications for enzyme engineering.

1 citation

experimental assay

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BiochemistryMolecular CellPeer-reviewedFeb 17, 2025

AcrVIB1 inhibits CRISPR-Cas13b immunity by promoting unproductive crRNA binding accessible to RNase attack

Katharina G. Wandera, Stefan Schmelz, Angela Migur…·Chase L. Beisel

This paper shows AcrVIB1 inhibits CRISPR-Cas13b by promoting unproductive crRNA binding and degradation, revealing a mechanism to suppress immune defense.

7 citations

CRISPR screenexperimental assay

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