Author

Lukas Englmaier

Austrian Academy of SciencesORCID 0000-0002-0082-2616Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Andreas VillungeriD· 2 shared Filip GallobiD· 2 shared Yevheniia Tyshchenko· 2 shared Cristián Eggers· 1 shared Danica DrpiciD· 1 shared Daniel MalzliD· 1 shared Dominika TučkováiD· 1 shared Felix EichiniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

Andreas VillungerORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMar 28, 2026

Inhibition of V-ATPase function drives apoptosis via GCN1/GCN2 kinase signaling

Filip Gallob, Severin Lechner, Dominika Tučková…·Andreas Villunger

The paper identifies V-ATPase inhibition as a trigger for apoptosis via GCN1/GCN2 kinase signaling and MCL-1 depletion, revealing a therapeutic target in certain cancers.

DOI 10.64898/2026.03.27.714872

experimental assaycancer & tumor microenvironment

𝕏 PostBluesky

Cell BiologybioRxivPreprintOct 24, 2025

tRNA thiolation defects disrupt cellular proteostasis and tissue homeostasis in mammals

Lukas Englmaier, Marta Walczak, Daniel Malzl…·Andreas Villunger

This study shows that tRNA thiolation defects in humans cause proteostasis and tissue homeostasis issues, but mice with similar defects develop normally.

DOI 10.1101/2025.10.24.684405

humanmouse

𝕏 PostBluesky