Author

Zhixiong Ma

Purdue University West LafayetteORCID 0000-0002-0088-8041Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Jingliang ZhangiD· 2 shared Muriel EatoniD· 2 shared Tiange XiaoiD· 2 shared William C. SkarnesiD· 2 shared Xiaoling CheniD· 2 shared Yang YangiD· 2 shared Zhuo HuangiD· 2 shared Anthony C. Park· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersNeuroscience

Jingliang ZhangORCID

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2 papers

Recent·Most cited

Severe deficiency of voltage-gated sodium channel NaV1.2 elevates neuronal excitability in adult mice

Jingliang Zhang, Xiaoling Chen, Muriel Eaton, Jiaxiang Wu, Zhixiong Ma, Shirong Lai, Anthony Park, Talha S. Ahmad, Zhefu Que, Ji Hea Lee, Tiange Xiao, Yuansong Li, Yujia Wang, Maria I. Olivero-Acosta, James A. Schaber, , Yang Yang

This study shows that severe NaV1.2 deficiency increases neuronal excitability in adult mice, challenging the current understanding of its role in autism and epilepsy.

Cell Reports · Aug 1, 2021Peer-reviewedNeuroscience

Characterization of a gene-trap knockout mouse model of Scn2a encoding voltage-gated sodium channel Nav1.2

Muriel Eaton, Jingliang Zhang, Zhixiong Ma, Anthony C. Park, Emma E Lietzke, Chloé Maricela Romero, Yushuang Liu, Emily Rose Coleman, Xiaoling Chen, Tiange Xiao, Zhuo Huang, William C. Skarnes, Wendy A. Koss, Yang Yang

This paper characterizes a Scn2a gene trap knockout mouse model with reduced Nav1.2 expression, showing behavioral and physiological abnormalities associated with Scn2a deficiency.

bioRxiv · Jun 23, 2020 · PreprintNeuroscience