Author

Agbonlahor Okhuarobo

Scripps Research InstituteORCID 0000-0002-0102-558XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

Candice ContetiD· 3 shared Catherine LopeziD· 3 shared Max KreifeldtiD· 3 shared Giovana C. MacedoiD· 2 shared Harpreet SidhuiD· 2 shared Jeffery L. DunningiD· 2 shared Marisa RobertoiD· 2 shared Michal BajoiD· 2 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersNeuroscience

Candice ContetORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Functional remodeling of the parasubthalamic nucleus drives alcohol drinking escalation in dependence

Jeffery L. Dunning, Max Kreifeldt, Agbonlahor Okhuarobo, Catherine Lopez, Florence P. Varodayan, Michal Bajo, R MAINS SMITH, Celeste Moreau, Colton Krull, Leeann Shu, Allison White, Giovana C. Macedo, Harpreet Sidhu, Howard C. Becker, Marisa Roberto, , Candice Contet

This study identifies PSTN Crh neurons as a key driver of alcohol drinking escalation in dependence, showing their role shifts from inhibitory to stimulatory with dependence.

bioRxiv · May 5, 2026 · PreprintNeuroscience

Mouse parasubthalamic Crh neurons drive alcohol drinking escalation and behavioral disinhibition

Max Kreifeldt, Agbonlahor Okhuarobo, Jeffery L. Dunning, Catherine Lopez, Giovana C. Macedo, Harpreet Sidhu, Candice Contet

This study identifies PSTN Crh neurons as drivers of alcohol drinking escalation and behavioral disinhibition in mice, independent of CRF1 signaling.

bioRxiv · Jul 6, 2024 · PreprintNeuroscience

Ethanol's interaction with BK channel α subunit residue K361 does not mediate behavioral responses to alcohol in mice

Agbonlahor Okhuarobo, Max Kreifeldt, Pauravi J. Gandhi, Catherine Lopez, Briana Martinez, Kiera Fleck, Michal Bajo, Pushpita Bhattacharyya, Alex M. Dopico, Marisa Roberto, Amanda J. Roberts, Gregg E. Homanics, Candice Contet

This study investigates whether ethanol's interaction with BK channel α subunit residue K361 mediates behavioral responses to alcohol in mice, finding no significant effects on ethanol-related behaviors or consumption.

Molecular Psychiatry · Dec 22, 2023Peer-reviewedNeuroscience