Author

Nwamaka J. Idigo

Wellcome Centre for Cell BiologyORCID 0000-0002-0723-3793Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Catherine M. AbbottiD· 1 shared Christina AmbrosiiD· 1 shared Christos SpanosiD· 1 shared Devisree ValsakumariD· 1 shared Dinesh C. SoaresiD· 1 shared Elana BryaniD· 1 shared Juri RappsilberiD· 1 shared Katy A. McLaughliniD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenetics

Catherine M. AbbottORCID

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2 papers

Recent·Most cited

Nucleosomal Asymmetry Shapes Histone Mark Binding and Promotes Poising at Bivalent Domains

Elana Bryan, Devisree Valsakumar, Nwamaka J. Idigo, Marie Warburton, Kimberly M. Webb, Katy A. McLaughlin, Christos Spanos, Simone Lenci, Viktoria Major, Christina Ambrosi, Simon Andrews, Tuncay Baubec, Juri Rappsilber, Philipp Voigt

The paper shows that asymmetric bivalent nucleosomes at developmental gene promoters promote a poised state by recruiting repressive H3K27me3 binders and specific chromatin proteins like KAT6B, which is critical for ESC differentiation.

Molecular Cell · Dec 27, 2024Peer-reviewedMolecular Biology

Translation elongation factor 1A2 (eEF1A2) is encoded by one of four closely related eef1a genes and is dispensable for survival in zebrafish

Nwamaka J. Idigo, Dinesh C. Soares, Catherine M. Abbott

The paper investigates the role of eEF1A2 in zebrafish by generating null mutations and compares phenotypes with morphants and mice, finding zebrafish can survive without eEF1A2.

Bioscience Reports · Jan 1, 2020Peer-reviewedGenetics