Author

Sana Intidhar Labidi‐Galy

University of GenevaORCID 0000-0002-0824-3475Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

Aditi GulatiiD· 1 shared Alberto ValdeolivasiD· 1 shared Andrew N.J. TuttiD· 1 shared Anna Moyseos· 1 shared Astrid Chanfon· 1 shared Christopher J. LordiD· 1 shared Demeter TúrósiD· 1 shared Dragomir B. KrasteviD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Aditi GulatiORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Spatiotemporal organisation of residual disease in mouse and human BRCA1-deficient mammary tumours and breast cancer

Demeter Túrós, Morgane Decollogny, Anna Moyseos, Astrid Chanfon, Myriam Siffert, Joanne Bousmar, Lou Romanens, Jean‐Christophe Tille, Olivier Trédan, Sana Intidhar Labidi‐Galy, Alberto Valdeolivas, Sven Rottenberg

This study maps the spatiotemporal organization of minimal residual disease in BRCA1-deficient mouse and human breast tumors, identifying a drug-tolerant EMT-like cell population linked to poor clinical outcomes.

Nature Communications · Jun 11, 2026Peer-reviewedCancer Biology

Reversion mutations define minimal BRCA1/2 requirements for therapy resistance

Lorena Magraner-Pardo, William Kerrison, Dragomir B. Krastev, Rachel Alcraft, Hui Xiao, Rachel Brough, Feifei Song, Sheyne S. A. Choi, Aditi Gulati, Manuel Jorge Rodrigues, Sana Intidhar Labidi‐Galy, Éric Pujade-Lauraine, Isabelle Laure Ray-Coquard, Syed Haider, Stephen J. Pettitt, , Christopher J. Lord

The paper identifies BRCA1/2 reversion mutations associated with drug resistance in cancer patients and highlights key domains critical for resistance.

bioRxiv · May 25, 2026 · PreprintCancer Biology