Author

Frida Belinky

National Institutes of HealthORCID 0000-0002-0857-0660Unclaimed ORCID profile · follow to get notified of new preprints

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:DNA methylation· 2 Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Wei WuiD· 2 shared Alonso Arana· 1 shared Ana Beatriz EnriqueziD· 1 shared André NussenzweigiD· 1 shared André NussenzweigiD· 1 shared Andre StanlieiD· 1 shared Aseem Z. AnsariiD· 1 shared Ashish A. SharmaiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersMolecular Biology

Wei WuORCID

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3 papers

Recent·Most cited

Neutralization activity in chronic HIV infection is characterized by a distinct programming of follicular helper CD4 T cells

Eirini Moysi, Ashish A. Sharma, Sijy J. O'Dell, Spiros Georgakis, Perla Mariana Del Rio Estrada, Khader Ghneim, Alonso Arana, Fernanda Torres-Ruiz, Mauricio González-Navarro, Yara Andrea Luna-Villalobos, Santiago Ríos, Gustavo Reyes‐Terán, Margaret H. Beddall, Sung Hee Ko, Frida Belinky, , Constantinos Petrovas

The paper identifies distinct follicular helper CD4 T cell programming in HIV-positive individuals who produce broadly neutralizing antibodies, linking it to improved B cell follicle architecture and reduced suppressor cells.

Journal of Clinical Investigation · Jul 14, 2026Peer-reviewedImmunology

Linking Dynamic DNA Secondary Structures to Genome Instability

Gabriel E. Matos-Rodrigues, Niek van Wietmarschen, Wei Wu, Veenu Tripathi, Natasha C. Koussa, Raphael Pavani, William J. Nathan, Frida Belinky, Ashraf Mohammed, Марек Напиерала, Karen Usdin, Aseem Z. Ansari, Sergei M. Mirkin, André Nussenzweig

The paper identifies dynamic DNA secondary structures in vivo that contribute to genome instability by analyzing ssDNA converted to DSBs using S1-END-seq.

bioRxiv · Jan 19, 2022 · PreprintMolecular Biology

The Dystonia Gene THAP1 Controls DNA Double Strand Break Repair Choice

Kenta Shinoda, Dali Zong, Elsa Callén, Wei Wu, Lavinia C. Dumitrache, Frida Belinky, Raj Chari, Nancy Wong, Momoko Ishikawa, Andre Stanlie, Trisha Multhaupt‐Buell, Nutan Sharma, Laurie J. Ozelius, Michelle E. Ehrlich, Peter J. McKinnon, André Nussenzweig

This study identifies a transcriptional network involving THAP1 that regulates SHLD1 expression, affecting DNA repair choice and linking DNA damage to dystonia.

Molecular Cell · Apr 15, 2021Peer-reviewedMolecular Biology