Author

Mathias A. Böhme

Leibniz-Forschungsinstitut für Molekulare PharmakologieORCID 0000-0002-0947-9172Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander M. WalteriD· 3 shared Anthony W. McCarthyiD· 2 shared Meida JusyteiD· 2 shared Ali Pejmun HaghighiiD· 1 shared Alix E. Bonard· 1 shared Andreas Till GrasskampiD· 1 shared Astrid G. PetzoldtiD· 1 shared Christine Brigitte BeuscheliD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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3 shared papersNeuroscience

Alexander M. WalterORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 16, 2022

Release site plasticity via Unc13A regulatory domains mediates synaptic short-term facilitation and homeostatic potentiation

Meida Jusyte, Natalie Blaum, Mathias A. Böhme…·Alexander M. Walter

This study identifies how Unc13A regulatory domains control synaptic plasticity by modulating release site participation in Drosophila synapses.

DOI 10.1101/2022.11.16.516613

Drosophilastatistical model

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NeurosciencebioRxivPreprintAug 26, 2020

Tetanus neurotoxin sensitive SNARE-mediated glial signaling limits motoneuronal excitability

Mathias A. Böhme, Anthony W. McCarthy, Monika Berezeckaja…·Alexander M. Walter

The paper identifies non-neuronal Synaptobrevin (Syb) as essential for glial function in Drosophila motoneurons, showing that disrupting Syb leads to nerve disintegration and paralysis.

DOI 10.1101/2020.08.26.268011

Drosophilaexperimental assay

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NeuroscienceNature CommunicationsPeer-reviewedMar 6, 2019

Rapid active zone remodeling consolidates presynaptic potentiation

Mathias A. Böhme, Anthony W. McCarthy, Andreas Till Grasskamp…·Alexander M. Walter

The paper identifies a molecular mechanism for presynaptic plasticity involving rapid remodeling of active zone modules, which sustains neurotransmitter release potentiation over time.

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