Anthony W. McCarthy

The paper shows that spontaneous neurotransmission at synapses predicts their responsiveness to action potentials in Drosophila NMJs.

This study shows that loss of basal forebrain cholinergic signaling leads to microglial activation and exaggerated inflammatory responses to systemic inflammation.

The paper identifies non-neuronal Synaptobrevin (Syb) as essential for glial function in Drosophila motoneurons, showing that disrupting Syb leads to nerve disintegration and paralysis.

The paper identifies a molecular mechanism for presynaptic plasticity involving rapid remodeling of active zone modules, which sustains neurotransmitter release potentiation over time.