Author

Marissa A. Menard

University of Alabama at BirminghamORCID 0000-0002-0976-3448Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Laura A. Volpicelli‐DaleyiD· 2 shared Charlotte F. BrzozowskiiD· 1 shared Douglas Narbert· 1 shared Eizo MarutaniiD· 1 shared Fumito IchinoseiD· 1 shared Harshita ChallaiD· 1 shared Hong Wang· 1 shared Hong‐Yuan ChuiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPhysiology

Laura A. Volpicelli‐DaleyORCID

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3 papers

Recent·Most cited

A second-generation ''hypoxia in a pill'' rescues neurodegenerative phenotypes across distinct mouse models

Hong Wang, Eizo Marutani, L. Zazzeron, Marissa A. Menard, Laura A. Volpicelli‐Daley, Fumito Ichinose, Vamsi Krishna Mootha

This paper shows that a second-generation hypoxia-in-a-pill treatment rescues neurodegenerative phenotypes in multiple mouse models.

Proceedings of the National Academy of Sciences · Oct 9, 2026Peer-reviewedPhysiology

α-Synuclein aggregates in corticostriatal terminals impair glutamatergic transmission in the absence of neurodegeneration

Charlotte F. Brzozowski, Zoe N. Fokakis, Marissa A. Menard, Harshita Challa, IGNACIO GALLARDO, J Dominic Hall, Douglas Narbert, Michael Millett, J. Andrew Hardaway, Mark S. Moehle, Laura A. Volpicelli‐Daley

This study shows that α-synuclein aggregates in corticostriatal terminals impair glutamatergic transmission without causing neurodegeneration.

bioRxiv · Aug 7, 2026 · PreprintNeuroscience

Impaired Functional Connectivity of Cortico-Amygdala Pathway Can Drive Social Behavior Deficits in Synucleinopathies

Wei Zhou, Samuel R. Daniels, Vijay Singh, Marissa A. Menard, Martha L. Escobar Galvis, Hong‐Yuan Chu

This study shows that -synuclein aggregation disrupts cortico-amygdala connectivity, leading to social behavior deficits in synucleinopathies, and that restoring this connection can rescue the behavior.

Neurobiology of Disease · Oct 13, 2024Peer-reviewedNeuroscience