Author

Marta Miret-Cuesta

Barcelona Institute of Science and TechnologyORCID 0000-0002-1083-5904Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Research Focus:gene regulatory network inference· 3 Organism:mouse· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

Manuel IrimiaiD· 3 shared Sophie BonnaliD· 3 shared L. IñigueziD· 2 shared Alfonsa Zamora‐MoratallaiD· 1 shared Álvaro Fernández‐BlancoiD· 1 shared Amaya López-PascualiD· 1 shared André GohriD· 1 shared Bárbara PernauteiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersNeuroscience

Manuel IrimiaORCID

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3 papers

Recent·Most cited

A highly conserved neuronal microexon in DAAM1 controls actin dynamics, RHOA/ROCK signaling, and memory formation

Patryk Poliński, Marta Miret-Cuesta, Alfonsa Zamora‐Moratalla, Federica Mantica, Gerard Cantero-Recasens, Carlotta Viana, Miguel Sabariego-Navarro, Davide Normanno, L. Iñiguez, Sophie Bonnal, Raúl Gómez-Riera, Hugo Fanlo-Ucar, Dominic S. Yap, María Martínez de Lagrán Cabredo, Álvaro Fernández‐Blanco, , Manuel Irimia

Repositori digital de la UPF (Universitat Pompeu Fabra) · Jan 1, 2025Peer-reviewedNeuroscience

Control of pancreatic islet function and glucose homeostasis by a novel microexon program misregulated in type 2 diabetes

Jonàs Juan‐Mateu, Simon Bajew, Marta Miret-Cuesta, L. Iñiguez, Amaya López-Pascual, Sophie Bonnal, Goutham Atla, Sílvia Bonàs‐Guarch, Jorge P. Ferrer, Juan Valcárcel, Manuel Irimia

The paper identifies a microexon program in pancreatic islets regulated by SRRM3, which affects insulin secretion and is linked to type 2 diabetes risk.

bioRxiv · Apr 2, 2022 · PreprintMolecular Biology

A developmentally programmed splicing failure attenuates the DNA damage response during mammalian zygotic genome activation

Christopher D. R. Wyatt, Bárbara Pernaute, André Gohr, Marta Miret-Cuesta, Lucía Goyeneche, Quirze Rovira, Ozren Bogdanović, Sophie Bonnal, Manuel Irimia

The paper shows that during zygotic genome activation, embryos exhibit temporary splicing failures in DNA damage response genes, regulated by Snrpb and Snrpd2, which reduce DNA damage response.

bioRxiv · Nov 25, 2020 · PreprintDevelopmental Biology