Author

Alişya A. Anlaş

University of PennsylvaniaORCID 0000-0002-1175-5164Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Dennis E. DischeriD· 2 shared Jason C. AndrechakiD· 1 shared Lawrence J. DoolingiD· 1 shared Mai WangiD· 1 shared Markus T. SprengeriD· 1 shared Maximilian Wang· 1 shared Michael P. TobiniD· 1 shared Nicholas M. Ontko· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBioengineering

Dennis E. DischerORCID

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2 papers

Recent·Most cited

Clustered macrophages cooperate to eliminate tumors via coordinated intrudopodia

Lawrence J. Dooling, Alişya A. Anlaş, Michael P. Tobin, Nicholas M. Ontko, Tristan Marchena, Maximilian Wang, Jason C. Andrechak, Dennis E. Discher

This study shows that macrophage clusters enhance tumor phagocytosis through coordinated intrudopodia extending between cancer cells.

Proceedings of the National Academy of Sciences · Jul 1, 2025Peer-reviewedBioengineering

Matrix stiffness induces heritable changes in chromosome numbers, consistent with solid tumor heterogeneity

Alişya A. Anlaş, Markus T. Sprenger, Mai Wang, Nick Ontko, Steven Phan, Dennis E. Discher

The paper shows matrix stiffness induces heritable chromosome changes in cells, linking mechanical stress to genetic instability in solid tumors.

bioRxiv · Jan 28, 2025 · PreprintBiophysics