Author

Alyssa Grogan

CVPath InstituteORCID 0000-0002-1505-3311Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Aloke Virmani FinniD· 2 shared Tatsuya ShirakiiD· 2 shared Aarohan M BurmaiD· 1 shared Alan Thomas RemaleyiD· 1 shared Amit Kumar DeyiD· 1 shared Ann Y. ParkiD· 1 shared Aran SoniD· 1 shared Atsuki EniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMolecular Biology

Aloke Virmani FinnORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologyCellPeer-reviewedOct 1, 2026

Immunoregulatory gene GIMAP6 suppresses lethal atherosclerotic vasculopathy and ischemic heart failure

Xiang Chen, Kaja S. Arusha, Danielle Springer…·Michael J. Lenardo

This study shows that GIMAP6 loss leads to inflammatory vasculopathy and atherosclerosis, causing heart failure and early death, and identifies GIMAP6 as a protective factor against cardiovascular disease.

DOI 10.1016/j.cell.2026.09.032

humanexperimental assay

𝕏 PostBluesky

Molecular BiologybioRxivPreprintApr 4, 2026

RNA polymerase loss by nuclear rupture drives LMNA cardiomyopathy

Atsuki En, Melanie E. Gucwa, Erjola Rapushi…·Kohta Ikegami

This study shows that nuclear rupture in LMNA-DCM leads to transcriptional deficiency and identifies resealing as a protective mechanism that is ultimately insufficient.

1 citation

mousehuman

𝕏 PostBluesky

ImmunologyAnnals of the Rheumatic DiseasesPeer-reviewedDec 13, 2025

Th9-endothelial cell crosstalk promotes inflammatory atherosclerotic cardiovascular disease

Ishita Baral, Yvonne Baumer, Aarohan M Burma…·Daniella M. Schwartz

This study identifies Th9 cells and IL-9R/STAT3 signaling as drivers of atherosclerosis in inflammatory conditions like psoriasis.

5 citations

mousehuman

𝕏 PostBluesky