Author

Ning Ma

Guangzhou LaboratoryORCID 0000-0002-1775-2992Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Ning Ma1 paper · Stanford Medicine

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Binrui JiiD· 1 shared Diyang Wang· 1 shared Gongcheng HuiD· 1 shared Guangjin PaniD· 1 shared Haidong WangiD· 1 shared Hanwen Wang· 1 shared Henghui Xu· 1 shared Hongjie YaoiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMolecular Biology

Binrui JiORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Vascularized Human Cardiac Organoids Reveal Endothelial-Cardiomyocyte Crosstalk and Mechanisms of Carfilzomib-Induced Cardiotoxicity

Yuan Yang, Liyang Zhao, Honglin Tao, Yao Zhang, Hanwen Wang, Henghui Xu, Ning Ma, Ning‐Yi Shao, Joe Z. Zhang

This study creates vascularized human cardiac organoids to study endothelial-cardiomyocyte interactions and cardiotoxicity mechanisms, finding bidirectional crosstalk and ER stress pathways involved in drug toxicity.

bioRxiv · Apr 16, 2026 · PreprintCell Biology

CTCF mutation at R567 causes developmental disorders via 3D genome rearrangement and abnormal neurodevelopment

Jie Zhang, Gongcheng Hu, Yuli Lu, Huawei Ren, Yin Huang, Yulin Wen, Binrui Ji, Diyang Wang, Haidong Wang, Huisheng Liu, Ning Ma, Lingling Zhang, Guangjin Pan, Yibo Qu, Hua Wang, , Hongjie Yao

This study shows that a CTCF R567W mutation causes developmental disorders by altering 3D genome structure and neurodevelopmental pathways in mouse and human models.

Nature Communications · Jul 1, 2024Peer-reviewedMolecular Biology