Author

Robert George Weiss

Johns Hopkins UniversityORCID 0000-0002-1844-8677Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Albert J. R. HeckiD· 1 shared Amin SabetiD· 1 shared An‐Chi WeiiD· 1 shared Anna V. LiiD· 1 shared Ashish K GuptaiD· 1 shared Eleonora Corradini· 1 shared Elizabeth D. LuczakiD· 1 shared Jiadi XuiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperMolecular Biology

Albert J. R. HeckORCID

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2 papers

Recent·Most cited

Mitochondrial Oxidative Phosphorylation Capacity in Skeletal Muscle Measured by Ultrafast Z-Spectroscopy (UFZ) MRI at 3T

Licheng Ju, Michael Schär, Kexin Wang, Anna V. Li, Yihan Wu, Thomas Jake Samuel, Sandeep Ganji, Peter C.M. van Zijl, Nirbhay Narayan Yadav, Robert George Weiss, Jiadi Xu

This study evaluates ultrafast Z-spectroscopy MRI with pH correction for measuring mitochondrial oxidative phosphorylation in human skeletal muscle.

Magnetic Resonance in Medicine · Oct 20, 2024Peer-reviewedBiophysics

Mitochondrial CaMKII causes metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy

Elizabeth D. Luczak, Yuejin Wu, Jonathan M. Granger, Mei-ling A. Joiner, Nicholas R. Wilson, Ashish K Gupta, Priya Umapathi, Kevin R. Murphy, Oscar E. Reyes Gaido, Amin Sabet, Eleonora Corradini, Wen-Wei Tseng, Yibin Wang, Albert J. R. Heck, An‐Chi Wei, , Mark E. Anderson

This study shows mitochondrial CaMKII activation leads to metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy, with potential therapeutic targets identified.

Nature Communications · Sep 4, 2020Peer-reviewedMolecular Biology