Author

Shan Li

California Institute of TechnologyORCID 0000-0002-2146-2453Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Shan Li2 papers · University of Chinese Academy of Sciences

Biology Disciplines

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Neuroscience· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Feng WangiD· 2 shared Tsui‐Fen ChouiD· 2 shared Andrew R. FindlayiD· 1 shared Andrey AndreeviD· 1 shared Arthur J. Y. HuangiD· 1 shared Brian P. HanrattyiD· 1 shared Brianna Garcia· 1 shared Chaodong ZhangiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersNeuroscience

Feng WangORCID

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3 papers

Recent·Most cited

stk32a links sleep homeostasis to suppression of sensory and motor systems

Steven Tran, Jasmine Emtage, Chaodong Zhang, Xiaoyao Liu, Marina Lecoeuche, Andrey Andreev, Grigorios Oikonomou, Sujatha Narayan, Brianna Garcia, Tasha Cammidge, Cristina Gonzales, Hannah Hurley, Misha Yap, Shan Li, Feng Wang, , David A. Prober

This study identifies stk32a as a conserved sleep regulator that links neuropeptidergic signaling to sleep homeostasis by suppressing sensory and motor systems in zebrafish and mice.

SLEEP · May 1, 2026Peer-reviewedNeuroscience

Defined cellular reprogramming of androgen receptor-active prostate cancer to neuroendocrine prostate cancer

Shan Li, Kai Song, Huiyun Sun, Yong Tao, Arthur J. Y. Huang, Vipul Bhatia, Brian P. Hanratty, Radhika A. Patel, Henry W. Long, Colm M. Morrissey, Michael C. Haffner, Peter S. Nelson, Thomas G. Graeber, John K. Lee

This study reprograms AR-active prostate cancer to NEPC using ASCL1 and NeuroD1, revealing their role in silencing AR and altering chromatin structure.

bioRxiv · Feb 12, 2025 · PreprintCancer Biology

DNAJB6 Isoform Specific Knockdown: Therapeutic Potential for LGMDD1

Andrew R. Findlay, May M. Paing, Jil Daw, Rocío Bengoechea, Sara K. Pittman, Shan Li, Feng Wang, Timothy M. Miller, Heather L. True, Tsui‐Fen Chou, Conrad Chris Weihl

This study investigates DNAJB6 isoform-specific knockdown as a potential therapy for LGMDD1, showing that reducing DNAJB6b levels in a mouse model corrected proteomic disease signatures.

bioRxiv · Nov 17, 2022 · PreprintNeuroscience