Author

Vanessa Brinkmann

Leibniz Institute of Environmental MedicineORCID 0000-0002-2284-3143Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Molecular Biology· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 3 Approach:experimental assay· 3 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alfonso SchiaviiD· 2 shared Natascia VenturaiD· 2 shared Alberto DelaidelliiD· 1 shared Alisa KahleriD· 1 shared Anna LaromaineiD· 1 shared Anne Hemmers· 1 shared Arash SamieiiD· 1 shared Charlotte EsseriD· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Alfonso SchiaviORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Molecular BiologyCell Death and DiseasePeer-reviewedJul 13, 2024

The eEF2 kinase coordinates the DNA damage response to cisplatin by supporting p53 activation

Jonathan K. M. Lim, Arash Samiei, Alberto Delaidelli…·Poul H. B. Sorensen

This study shows eEF2K is critical for the DNA damage response to cisplatin, promoting p53 activation and DNA repair depending on damage severity.

13 citations

humanC. elegans

𝕏 PostBluesky

NeuroscienceNature CommunicationsPeer-reviewedMay 12, 2022

Neuroligin-mediated neurodevelopmental defects are induced by mitochondrial dysfunction and prevented by lutein in C. elegans

Silvia Maglioni, Alfonso Schiavi, Marlen Melcher…·Natascia Ventura

This study identifies lutein as a potential therapeutic for mitochondrial dysfunction-induced neurodevelopmental defects in C. elegans by restoring neuroligin expression.

30 citations

C. elegansexperimental assay

𝕏 PostBluesky

Molecular BiologyAntioxidantsPeer-reviewedMar 23, 2022

Aryl hydrocarbon receptor-dependent and -independent pathways mediate curcumin anti-aging effects

Vanessa Brinkmann, Margherita Romeo, Lucie Larigot…·Natascia Ventura

This study investigates how curcumin's anti-aging effects are mediated through AhR-dependent and -independent pathways in different contexts.

14 citations

C. eleganshuman

𝕏 PostBluesky