Author

Matthias Preuße

Helmholtz Centre for Infection ResearchORCID 0000-0002-2775-3139Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

Aleksei AgapoviD· 1 shared Anna OlinaiD· 1 shared Christian HackeriD· 1 shared Edze Rients WestraiD· 1 shared Maria Garcia Castillo· 1 shared Maureen MicalettoiD· 1 shared Nicolas Oswaldo GomeziD· 1 shared Rafael CantóniD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMicrobiology

Aleksei AgapovORCID

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1 paper

Recent·Most cited

Loss of restriction-modification methyltransferases drives persistence to fluoroquinolones in Pseudomonas aeruginosa

Maureen Micaletto, Nicolas Oswaldo Gomez, Anna Olina, Aleksei Agapov, Urszula Łapińska, Matthias Preuße, Maria Garcia Castillo, Christian Hacker, Rafael Cantón, Stineke van Houte, Susanne Häussler, Edze Rients Westra, Stefano Pagliara

This study shows that loss of type I restriction-modification methyltransferases in Pseudomonas aeruginosa increases fluoroquinolone persistence through enhanced SOS response and slower growth.

bioRxiv · Sep 26, 2026 · PreprintMicrobiology