Author

Miki Jishage

New York UniversityORCID 0000-0002-2835-2564Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Maxwell McDermottiD· 2 shared Stefan FeskeiD· 2 shared Abdelaziz Belkadi· 1 shared Adam C. StraubiD· 1 shared Ahmed Emam AbdelnabyiD· 1 shared Aidan T. PezackiiD· 1 shared Amal Taher ElhawiD· 1 shared Anthony Y. TaoiD· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

Maxwell McDermottORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Orai1α and Orai1β more potently promote NFAT activation and transcription than Orai2 and Orai3

Ahmed Emam Abdelnaby, Yin‐Hu Wang, Cory J. Benson, Isani Paolo Perez, Shen Mark, Maxwell McDermott, Miki Jishage, Amal Taher Elhaw, Silvia Cruz‐Rangel, Raphael J Courjaret, Abdelaziz Belkadi, Fang Yu, Douglas S Prado, Shuai Yuan, Ping Xin, , Mohamed Trebak

The paper compares the ability of different Orai isoforms to promote NFAT activation and transcription, finding Orai1α and Orai1β are more potent than Orai2 and Orai3.

bioRxiv · Oct 23, 2026 · PreprintCell Biology

CTR1-mediated copper uptake orchestrates metabolic-epigenetic regulation of pathogenic TH17 cells in autoimmune disease

Lucile Noyer, Liwei Wang, Miki Jishage, Maxwell McDermott, Aidan T. Pezacki, Anthony Y. Tao, Ravichandran Ramasamy, Matthias Stadtfeld, Christopher J. Chang, Stefan Feske

This study shows that CTR1-mediated copper uptake is essential for pTH17 cell differentiation and function in autoimmune disease by linking copper homeostasis to metabolic and epigenetic regulation.

bioRxiv · Sep 28, 2026 · PreprintImmunology

Transcriptional down-regulation of metabolic genes by Gdown1 ablation induces quiescent cell re-entry into the cell cycle

Miki Jishage, Keiichi Ito, Chi-Shuen Chu, Xiaoling Wang, Masashi Yamaji, Robert G. Roeder

This study shows that Gdown1 ablation reduces Pol II recruitment to metabolic genes, leading to cell cycle re-entry and p53-dependent changes in hepatocytes.

Genes & Development · Jun 1, 2020Peer-reviewedMolecular Biology