Author

Giuseppe Alvise Ramirez

Vita-Salute San Raffaele UniversityORCID 0000-0002-2889-366XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:experimental assay· 1 Assay:flow cytometry· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Angelo Andrea ManfrediiD· 1 shared Annalisa CapobiancoiD· 1 shared Antonella Monno· 1 shared Augusto VaglioiD· 1 shared Barbara Rewerska· 1 shared Benjamin TerrieriD· 1 shared Chiara BaldiniiD· 1 shared Chris WallaceiD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

Angelo Andrea ManfrediORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Neutrophil Reprogramming Underlie Vasculopathy and Lung Disease in Systemic Sclerosis

Norma Maugeri, Giuseppe Alvise Ramirez, Annalisa Capobianco, Antonella Monno, Erika Arosio, Marco D’Ercole, Marco E. Bianchi, Patrizia Querini Rovere, Florenzo Iannone, Marco Matucci‐Cerinic, Angelo Andrea Manfredi

This study identifies neutrophil reprogramming in systemic sclerosis (SSc) driven by HMGB1+ extracellular vesicles, leading to lung inflammation and fibrosis, and suggests therapeutic targeting of HMGB1.

Cell Death and Disease · Apr 4, 2026Peer-reviewedImmunology

Genetically distinct clinical subsets, and associations with asthma and eosinophil abundance, within Eosinophilic Granulomatosis with Polyangiitis

Paul Lyons, James E. Peters, Federico Alberici, James Liley, Richard Coulson, William John Astle, Chiara Baldini, Francesco Bonatti, Maria Cinta Cid, Heather Elding, Giacomo Emmi, Jörg T. Epplen, Loı̈c Guillevin, David R.W. Jayne, Tao Jiang, , Kenneth G. C. Smith

This study identifies genetic subsets of EGPA associated with asthma and eosinophil levels, revealing two distinct clinical and genetic syndromes.

bioRxiv · Dec 10, 2018 · PreprintGenetics