Author

Marco Matucci‐Cerinic

Vita-Salute San Raffaele UniversityORCID 0000-0002-9324-3161Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Aleix Rius RigauiD· 1 shared Alexandru‐Emil MateiiD· 1 shared Andrea-Hermina Gyoerfi· 1 shared Angelo Andrea ManfrediiD· 1 shared Annalisa CapobiancoiD· 1 shared Antonella Monno· 1 shared Ayla Nadja StütziD· 1 shared Bilgesu Şafak TümerdemiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBioinformatics

Aleix Rius RigauORCID

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Author profile →

2 papers

Recent·Most cited

Spatial proteomic profiling of cardiac stromal populations and microenvironments in systemic sclerosis-associated primary myocardial involvement

Ayla Nadja Stütz, Giacomo Davide De Luca, Alexandru‐Emil Matei, Yinan Li, Tim Filla, Veronica Batani, Bilgesu Şafak Tümerdem, Aleix Rius Rigau, Cosimo Bruno, Wolfgang Merkt, Elric Zweck, Maike Buettner-Herold, Stefania Rizzo, Giovanni Peretto, Monica De Gaspari, , Andrea-Hermina Gyoerfi

The paper uses spatial proteomic profiling to analyze cardiac stromal populations and microenvironments in systemic sclerosis-associated myocarditis, identifying distinct fibroblast subtypes and their spatial interactions.

bioRxiv · Oct 19, 2026 · PreprintBioinformatics

Neutrophil Reprogramming Underlie Vasculopathy and Lung Disease in Systemic Sclerosis

Norma Maugeri, Giuseppe Alvise Ramirez, Annalisa Capobianco, Antonella Monno, Erika Arosio, Marco D’Ercole, Marco E. Bianchi, Patrizia Querini Rovere, Florenzo Iannone, Marco Matucci‐Cerinic, Angelo Andrea Manfredi

This study identifies neutrophil reprogramming in systemic sclerosis (SSc) driven by HMGB1+ extracellular vesicles, leading to lung inflammation and fibrosis, and suggests therapeutic targeting of HMGB1.

Cell Death and Disease · Apr 4, 2026Peer-reviewedImmunology