Author

Jason M. Karch

Baylor College of MedicineORCID 0000-0002-3023-7469Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Anna NozzaiD· 1 shared Arielys MendozaiD· 1 shared Daniel RamireziD· 1 shared Dexter J. RobichauxiD· 1 shared Dobromir DobreviD· 1 shared Donald M. BersiD· 1 shared Feng XiongiD· 1 shared Florian Bruns· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPathology

Anna NozzaORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJun 13, 2025

Atad3 is Essential for Mitochondrial Permeability Transition Pore Opening and Cardiac Ischemia Reperfusion Injury.

Dexter J. Robichaux, Sual J Lopez, Arielys Mendoza…·Jason M. Karch

This study identifies ATAD3 as an essential component of the mitochondrial permeability transition pore (mPTP), showing its role in mPTP opening and reducing cardiac ischemia reperfusion injury.

3 citations

mouseexperimental assay

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PathologybioRxivPreprintJan 29, 2025

Time-dependent Mitochondrial Remodeling in Experimental Atrial Fibrillation and Potential Therapeutic Relevance

Xiaoyan Qi, Feng Xiong, Jiening Xiao…·Stanley Nattel

This study examines mitochondrial changes during atrial fibrillation in animals and humans, showing early hyperfunction and damage, and demonstrates that mitochondrial-targeted therapy can prevent adverse remodeling and AF sustainability.

1 citation

humanmouse

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