William D. Lubell

This study designs a peptidomimetic inhibitor that blocks Vibrio cholerae adhesin FrhA, showing nanomolar binding affinity and reduced biofilm formation.

This study uses computational methods to analyze how CD36 phosphorylation alters its structure, affecting TSP-1 binding and fatty acid uptake.

This study shows that nonfibrillar Aβ aggregates in APPE693Q mice disrupt presynaptic function without causing inflammation.

This study shows that the CD36 modulator MPE-298 reduces infarct size and LCFA accumulation in mice after myocardial ischemia-reperfusion.

The paper identifies a tripeptide antagonist that blocks Vibrio cholerae adhesin FrhA with higher affinity than a previous pentapeptide ligand.