Author

Francisco Borrego

IkerbasqueORCID 0000-0002-3105-981XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Laura AmoiD· 2 shared Ainara Lopez-PardoiD· 1 shared Ainhoa Amarilla-Irusta· 1 shared Aitor Zarandona-GaraiiD· 1 shared Ane OrrantiaiD· 1 shared Aritz Tijero· 1 shared Arrate SevillaiD· 1 shared Bárbara Manzanares-Martín· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Laura AmoORCID

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2 papers

Recent·Most cited

Dual role of GABAB receptor in oligodendrocyte function and immune modulation in experimental multiple sclerosis

Laura Bayón‐Cordero, Blanca Isabel Ochoa-Bueno, Irene Luengas-Escuza, Rodrigo Senovilla-Ganzo, Izaskun Buendía, Chao Zheng, Eneritz Agirre, Gonçalo Castelo‐Branco, Laura Amo, Francisco Borrego, Fernando García‐Moreno, Frank Kirchhoff, Xianshu Bai, Carlos Matute, María Victoria Sánchez‐Gómez

This study shows GABAB receptors have dual roles in oligodendrocyte function and immune modulation in experimental MS, with baclofen showing therapeutic potential in reducing disease severity and promoting remyelination.

bioRxiv · Sep 9, 2025 · PreprintNeuroscience

Activated NK cells with a predominance of inhibitory receptors and a decidual-like phenotype expand after autologous HSCT in children with tumors.

Gabirel Astarloa-Pando, Diego Polanco-Alonso, Víctor Sandá, Ane Orrantia, Ainhoa Amarilla-Irusta, Sílvia Pérez-Fernández, Raquel Pérez-Garay, Arrate Sevilla, Ainara Lopez-Pardo, Aritz Tijero, Jose Javier Uriz, Mercedes Rey, Itziar Astigarraga, Bárbara Manzanares-Martín, Aitor Zarandona-Garai, , Francisco Borrego

The paper characterizes NK cell changes after autologous HSCT in children, finding an activated, decidual-like phenotype linked to cytokine levels and relapse risk.

Communications Medicine · May 28, 2025Peer-reviewedImmunology