An A. Vo

This study identifies MLT-11 as essential for C. elegans embryogenesis and shows conserved sequences in MLT-11 are critical for cuticle structure.

This study identifies mlt-11 as an NHR-23 target gene essential for C. elegans cuticle structure and function, showing its role in patterning cuticle layers and maintaining barrier integrity.

This paper shows that NHR-23 is essential for C. elegans development and apical extracellular matrix structure by regulating gene expression in seam and hypodermal cells.

The paper optimizes experimental parameters and provides new reagents for studying C. elegans lysosomal proteins, addressing challenges like protein degradation and detection.

The paper identifies distinct gene regulation by NHR-23 and SPE-44 during C. elegans spermatogenesis and shows their combined depletion affects additional genes.

The paper introduces the mIAA7 degron, which enhances auxin-mediated protein degradation efficiency in C. elegans, with some improvements in leakiness when combined with the C.e.AID2 system.

This study identifies MLT-11, a protease inhibitor essential for C. elegans molting and embryogenesis, showing its role in aECM remodeling and cuticle integrity.