Caterina Montani

This study shows that endogenous oxytocin alters brain network dynamics, increasing salience-related network states and decreasing sensory-dominated states in mice.

This study identifies sephin1's mechanism of action in proteostasis disruption by showing it binds G-actin, causes cytoskeleton misfolding, and modulates autophagy through mTORC1-TFEB and GCN2-eIF2-CHOP pathways.

This study shows that a transient developmental excitation-inhibition imbalance causes lasting social brain circuit dysfunction in mice, with behavioral and transcriptional consequences.

This study investigates developmental dysconnectivity in 22q11.2 deletion syndrome using a cross-species approach, finding age-specific fMRI connectivity changes with synaptic origins.

The paper identifies hypo- and hyperconnectivity subtypes in autism using cross-species fMRI, linking them to distinct biological mechanisms in mice and humans.

This study shows that Ube3a gene overdosage causes sex-biased effects on brain connectivity, behavior, and gene expression in mice, linking it to autism-related phenotypes.

This paper investigates brain network and behavioral changes in a mouse model of CTD and shows that perinatal gene therapy can partially reverse some symptoms but not all.

This study investigates how xanomeline, an M1/M4 agonist, affects brain functional connectivity in mice using phMRI, rsfMRI, and qEEG, showing both direct and modulatory effects on neural activity and connectivity.

This study examines how acute and repeated intranasal oxytocin administration affects brain-wide functional connectivity in mice, revealing distinct patterns of neural reconfiguration with potential clinical implications.