Author

Hirotake Ichise

Institute of Medical Science, The University of TokyoORCID 0000-0002-3424-6637Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Aki MinodaiD· 1 shared Boksik ChaiD· 1 shared Chung Chau Hon· 1 shared Constantinos Marios MikelisiD· 1 shared Dongwon Choi· 1 shared Georg A. HolländeriD· 1 shared Hiroto Ishii· 1 shared Hong CheniD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

Aki MinodaORCID

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 6, 2025

ASCL1 primes medullary thymic epithelial cell programs required for central tolerance

Nobuko Akiyama, Kenta Horie, Kano Namiki…·Taishin Akiyama

The paper identifies ASCL1 as a key regulator of mTEC function and central tolerance by analyzing single-cell RNA sequencing data from thymoma patients and mouse models.

DOI 10.1101/2025.08.06.668799

mousescRNA-seq

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Developmental BiologyJCI InsightPeer-reviewedJun 16, 2020

S1PR1 regulates the quiescence of lymphatic vessels by inhibiting laminar shear stress-dependent VEGF-C signaling

Xin Geng, Keisuke Yanagida, Racheal Grace Akwii…·R. Sathish Srinivasan

This study identifies S1PR1 as a regulator of lymphatic vessel quiescence by inhibiting LSS-dependent VEGF-C signaling in lymphatic endothelial cells.

62 citationstop 4%

mouseexperimental assay

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