Author

Seiji Shioda

Hoshi UniversityORCID 0000-0002-4164-6743Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Ari W. SchaleriD· 1 shared Atsuko NagaokaiD· 1 shared Avery M. RunyaniD· 1 shared Haruo OkadoiD· 1 shared Helen Y. Figueroa· 1 shared Hideki MiwaiD· 1 shared Hiroko ShimboiD· 1 shared Hirooki YabeiD· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Ari W. SchalerORCID

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2 papers

Recent·Most cited

High Sucrose Diets Contribute to Brain Angiopathy with Impaired Glucose Uptake, and Psychosis-related Higher Brain Dysfunctions in Mice

Shinobu Hirai, Hideki Miwa, Tomoko Tanaka, Kazuya Toriumi, Yasuto Kunii, Hiroko Shimbo, Takuya Sakamoto, Mizuki Hino, Ryuta Izumi, Atsuko Nagaoka, Hirooki Yabe, Tomoya Nakamachi, Seiji Shioda, Takashi Dan, Toshio Miyata, , Haruo Okado

This study shows that high-sucrose diets in Glo1-deficient mice cause brain angiopathy, impaired glucose uptake, and psychosis-related behaviors, with aspirin offering protection.

Science Advances · Nov 10, 2021Peer-reviewedNeuroscience

GPCR-mediated clearance of tau in post-synaptic compartments attenuates tau pathology in vivo

Ari W. Schaler, Avery M. Runyan, Stephanie L. Fowler, Helen Y. Figueroa, Seiji Shioda, Ismael Santa‐María, Karen Duff, Natura Myeku

This study shows that targeting GPCRs to enhance synaptic proteasome activity reduces tau accumulation and cognitive decline in a mouse model of Alzheimer's disease.

bioRxiv · Jan 21, 2020 · PreprintNeuroscience