Author

Marcus Henricsson

University of GothenburgORCID 0000-0002-4202-0339Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Organism:C. elegans· 3 Approach:experimental assay· 3 Research Focus:development & morphogenesis· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Jan BoréniD· 3 shared Marc PiloniD· 3 shared August QvistiD· 2 shared Delaney KaperiD· 2 shared Uroš RadovićiD· 2 shared Alba CarrerasiD· 1 shared Albin Stjernman· 1 shared Arash HaghikiaiD· 1 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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3 shared papersCell Biology

Jan BorénORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A pcyt-1 Allelic Series Reveals In Vivo Consequences of Reduced Phosphatidylcholine Synthesis in C. elegans

August Qvist, Delaney Kaper, Marcus Henricsson, Albin Stjernman, Jan Borén, Marc Pilon

This study characterizes the effects of reduced phosphatidylcholine synthesis in C. elegans using pcyt-1 mutant alleles, revealing compensatory lipid remodeling and specific developmental vulnerabilities.

G3 Genes Genomes Genetics · Jul 4, 2026Peer-reviewedCell Biology

Forward Genetics Identifies ptr-18 and Other Genes as Developmental Regulators of the Four-Cell Tail Tip in C. elegans

Uroš Radović, Marcus Henricsson, Jan Borén, Marc Pilon

bioRxiv · Aug 20, 2025 · PreprintCell Biology

Forward genetics in C. elegans reveals genetic adaptations to polyunsaturated fatty acid deficiency

Delaney Kaper, Uroš Radović, Per‐Olof Bergh, August Qvist, Marcus Henricsson, Jan Borén, Marc Pilon

This study identifies genetic suppressors of PUFA deficiency in C. elegans, showing that increasing PUFA levels, not stress responses, alleviates the defect.

eLife · Jul 8, 2025Peer-reviewedCell Biology

Gut microbial metabolite imidazole propionate impairs endothelial cell function and promotes the development of atherosclerosis

Vanasa Nageswaran, Alba Carreras, Leander Reinshagen, Katharina R. Beck, Jakob Steinfeldt, Marcus Henricsson, Pegah Ramezani Rad, Lisa Peters, Elisabeth Tamara Strässler, J.J. Lim, Barbara J. H. Verhaar, Yvonne Döring, Christian Weber, Maximilian König, Elisabeth Steinhagen‐Thiessen, , Arash Haghikia

This study shows that gut microbial metabolite imidazole propionate impairs endothelial function and promotes atherosclerosis in both human and animal models.

Arteriosclerosis Thrombosis and Vascular Biology · Mar 27, 2025Peer-reviewedSystems Biology