Author

Jonathan M. Weiss

Frederick National Laboratory for Cancer ResearchORCID 0000-0002-4467-5611Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:electronic health records· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

Daniel W. McVicariD· 2 shared Duncan E. DonohueiD· 2 shared Esta SterneckiD· 2 shared Kuppusamy BalamuruganiD· 2 shared Lois McKennettiD· 2 shared Shikha SharaniD· 2 shared Ariana VitaleiD· 1 shared Brad A. GoukeriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

Daniel W. McVicarORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 6, 2026

Metabolic maintenance of breast cancer cells and metastases through E-cadherin/YAP-dependent pyruvate carboxylase expression

Kuppusamy Balamurugan, Jonathan M. Weiss, Lois McKennett…·Esta Sterneck

This study identifies E-cadherin/YAP-dependent pyruvate carboxylase expression as a metabolic driver of breast cancer progression and metastasis, with therapeutic implications for epithelial TNBC.

DOI 10.64898/2026.04.13.718309

humanbulk RNA-seq

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Cancer BiologyCancer Research CommunicationsPeer-reviewedFeb 18, 2026

A simple liquid 3D cell culture paradigm models oxidative mitochondrial metabolism of epithelial breast cancer cells with relevance for lung metastases.

Kuppusamy Balamurugan, Melissa R. Mikolaj, Jonathan M. Weiss…·Esta Sterneck

This paper compares two 3D culture models for breast cancer cells, finding that the EmC model better mimics lung metastasis by altering metabolism and gene expression.

1 citation

humanproteomics

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