Martien J. Kas

This study replicates and extends findings linking social dysfunction to reduced default mode network connectivity in schizophrenia, Alzheimer's disease, and major depressive disorder.

This study shows that Pcdh9 deletion in mice causes synaptic overgrowth and excitatory transmission defects in the hippocampus, revealing its role in synaptic morphology and function.

This study assesses the robustness of Irwin tests across multiple sites for detecting drug-induced neurological effects in mice, finding reduced variability after protocol harmonization.

This study identifies CNTN4's role in cortical development and shows its interaction with APP affects neural elongation and spine density.

This study shows that sleep deprivation does not cause memory loss but suboptimal storage, which can be retrieved using optogenetics or roflumilast.

This study develops and validates an EEG-based method to measure visual deviance detection in freely behaving mice using light intensity changes as stimuli.

This study shows that sleep deprivation reduces specific spine types on certain dendritic branches in CA1 neurons, but not others, suggesting branch-specific effects on structural plasticity.

The paper estimates genetic correlations between eating disorder and substance-use-related phenotypes using GWAS data, finding both positive and negative associations.

This study identifies genetic variants associated with sociability as a continuous trait and finds genetic correlations with schizophrenia, major depressive disorder, and autism spectrum disorders.

This study investigates the shared genetic basis of anorexia nervosa and obsessive-compulsive disorder using a cross-disorder GWAS, finding a significant genetic correlation and associations with psychiatric and metabolic traits.

This paper uses transcriptomic imputation to identify gene-tissue associations linked to Anorexia Nervosa risk, finding significant links in both brain and metabolic tissues.