Author

Ronald P.C. Wong

Institute of Molecular BiologyORCID 0000-0002-4652-514XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Helle D. UlrichiD· 2 shared Aisha A. Kahloon· 1 shared Alexander LoeweriD· 1 shared Felizitas F. StiehleriD· 1 shared Fridolin KielischiD· 1 shared Hans-Peter WollscheidiD· 1 shared Ivan MikičićiD· 1 shared Jie Debbie Shi· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCell Biology

Helle D. UlrichORCID

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2 papers

Recent·Most cited

The nuclear actin cytoskeleton supports DNA double-strand break repair via VCP-mediated extraction of the KU70/80 complex from damaged chromatin

Kristine Hauschulte, Jie Debbie Shi, Ronald P.C. Wong, Felizitas F. Stiehler, Sebastian Richenhagen, Aisha A. Kahloon, Lea-Marie Vogts, Markus S Schraft, Tina Strauch, Stephanie Nick, Ivan Mikičić, Márton Gelléri, Fridolin Kielisch, Vassilis Roukos, Krishnaraj Rajalingam, , Hans-Peter Wollscheid

This study identifies Myo6's role in DSB repair by interacting with KU70/80 and VCP, facilitated by F-actin, to extract the KU70/80 complex from chromatin.

bioRxiv · Sep 2, 2026 · PreprintCell Biology

Post-replicative lesion processing limits DNA damage-induced mutagenesis

Katarzyna H. Masłowska, Ronald P.C. Wong, Helle D. Ulrich, Vincent Pagès

This study examines how the timing of lesion bypass during DNA replication affects mutagenesis, showing that TLS at certain lesions occurs post-replicatively and competes with DA, reducing mutagenicity.

Nucleic Acids Research · Mar 3, 2025Peer-reviewedMolecular Biology