Michael J. Schmeißer

This study investigates the effects of modulating VIP+ neurons in mouse corticolimbic regions on anxiety, social behavior, and fear learning, finding region-specific impacts on fear learning in the mPFC.

This study shows that low-dose radiation increases cortical excitability in mice, suggesting potential therapeutic use for neuronal circuit disorders.

This study shows that deleting glucocorticoid receptors in postnatal oligodendrocyte precursor cells reduces oligodendrocyte density, alters hippocampal activity, and impairs memory in adult mice.

The paper identifies early brain state maladaptations in mouse and human neuroinflammation that contribute to disease progression independent of relapse activity.

This study shows that deleting Shank3 in glutamatergic neurons of the anterior insular cortex causes autism-like behaviors and circuit dysfunction in mice.

This study shows that restoring CB1R in the anterior insular cortex of CB1R-deficient mice normalizes social novelty discrimination and reduces anxiety-like behavior without affecting other behaviors.

This study investigates how Neuroligin-2 interacts with MDGA1 and MDGA2 to regulate gephyrin aggregation and GABAAR function in mouse hippocampal synapses, revealing MDGA1's role in modulating these processes.

This study shows that early life stress alters the transcriptome of NG2+ glia, increases Nav channel activity, and impairs cognitive performance in adulthood.

This study identifies NECAB2's role in an endosomal pathway for mitochondrial stress response and repair in striatal synapses, linking its loss to synaptic loss and behavioral deficits.