Audrey T. Medeiros

The paper identifies DEAF1 as a repressor of synaptic genes, showing its role in limiting synaptogenesis and linking it to intellectual disability.

The paper investigates how VGCC abundance and spatial organization influence synaptic function in Drosophila motor neurons with different release probabilities.

This study examines the synaptic effects of α-synuclein-112, showing it impairs vesicle recycling through enhanced phospholipid binding and oligomerization.

This paper shows that excess α-synuclein impairs synaptic vesicle recycling by sequestering Hsc70, and that increasing Hsc70 levels can reverse this defect in a vertebrate synapse.