Author

John Jia En Chua

Agency for Science, Technology and ResearchORCID 0000-0002-5615-1014Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Adam C. PuchéiD· 1 shared Alexandros PoulopoulosiD· 1 shared Andrea J. RomanowskiiD· 1 shared Annarita PatriziiD· 1 shared Bekir AltasiD· 1 shared Cheryl BrandenburgiD· 1 shared Colin D. RobertsoniD· 1 shared Dilja Krueger‐BurgiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Adam C. PuchéORCID

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2 papers

Recent·Most cited

Phosphorylation Determines Whether Neuroligin-3 is at Excitatory or Inhibitory Synapses in Different Regions of the Brain

Bekir Altas, Liam P. Tuffy, Annarita Patrizi, Kalina P. Dimova, Tolga Soykan, Cheryl Brandenburg, Andrea J. Romanowski, Julia Whitten, Colin D. Robertson, Saovleak N. Khim, Garrett W. Crutcher, Mateusz C. Ambrozkiewicz, Oleksandr Yagensky, Dilja Krueger‐Burg, Matthieu Hammer, , Alexandros Poulopoulos

Biological Psychiatry · Dec 26, 2023Peer-reviewedNeuroscience

FEZ1 participates in human embryonic brain development by modulating neuronal progenitor subpopulation specification and migration

Yinghua Qu, Jonathan Jun-Yong Lim, Ömer An, Henry He Yang, Yi‐Chin Toh, John Jia En Chua

This study shows FEZ1 is essential for human brain development by regulating neuronal progenitor subpopulations and migration, with its deletion causing abnormalities in cortical layer formation.

iScience · Nov 20, 2023Peer-reviewedNeuroscience