Mathias Müsken

This study shows that Arp2/3 complex-dependent actin remodeling is necessary for efficient RSV uncoating in A549 cells, as Arp2 knockout cells exhibit reduced infection and viral mRNA expression.

This study reveals that ancestral Rhizobiaceae bacteria evolved photosynthesis in association with algae and plants, later losing it in some lineages that became plant symbionts or pathogens.

This study identifies an auxin transporter in rhizobial extracellular vesicles that enhances legume symbiosis by modulating auxin levels in the peribacteroid space.

This study identifies ScSOF, a fibronectin-binding surface protein of Streptococcus canis, as critical for endothelial adhesion and virulence through TraDIS screening.

The paper identifies three large virulence-factor-like proteins in a bacterial symbiosis, showing their transfer between species and potential roles in promoting cell contact and facilitating protein injection.

This study characterizes a phage that infects Nitrosomonas europaea, showing it disrupts membrane integrity and autotrophic metabolism, leading to host cell lysis.

The paper identifies virulent phage genomes in bacterial assemblies, challenging the assumption that virulent phages cannot persist in bacterial cells.

This study shows that glial cells, particularly astrocytes, promote HPAIV H7N7 infection in vitro by acting as primary virus producers, with microglia initiating pro-inflammatory responses.

This paper develops and characterizes a 2D porcine colonic organoid model that mimics native colon tissue for studying intestinal physiology and barrier function.

This study uses single-molecule microscopy to investigate influenza A virus-induced nanoscale cellular changes at the virus-cell interface, revealing receptor dynamics and actin cortex responses.

The paper isolates and characterizes four new Streptomyces phages with varying host ranges and temperate/virulent properties, expanding the available phage resources for studying antiviral strategies in Streptomyces.

This study identifies the Arp2/3 complex as critical for maintaining mast cell survival and tissue residency through actin-dependent mechano-protection.