Author

Eleanor J. McKay

University of EdinburghORCID 0000-0002-5690-6042Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:metabolomics· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Ami Onishi· 2 shared Ineke H. N. LuijteniD· 2 shared Robert K. SempleiD· 2 shared David B. SavageiD· 1 shared Jake Peter ManniD· 1 shared Jana Buchan· 1 shared Tore BengtssoniD· 1 shared Ula Kibildyte· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 31 mutual co-author links

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2 shared papersPhysiology

Ami Onishi

Mutual co-authors in cluster

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2 papers

Recent·Most cited

PhysiologyMolecular MedicinePeer-reviewedAug 4, 2025

Evaluation of Candidate ''Kill or Cure'' Strategies to Treat MFN2-related Lipodystrophy

Ineke H. N. Luijten, Xiong Weng, Ula Kibildyte…·Robert K. Semple

This study evaluates 'kill or cure' strategies for MFN2-related lipodystrophy, finding that ethanol mildly worsens the condition in mice while rapamycin is well-tolerated but lacks efficacy in targeting Mfn2R707W cells.

DOI 10.1186/s10020-025-01314-2

mouseproteomics

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PhysiologyAmerican Journal of Physiology-Endocrinology and MetabolismPeer-reviewedMar 28, 2025

The metabolically protective energy expenditure increase of Pik3r1-related insulin resistance is not explained by Ucp1-mediated thermogenesis

Ineke H. N. Luijten, Ami Onishi, Eleanor J. McKay…·Robert K. Semple

This study investigates whether increased energy expenditure in Pik3r1-related insulin resistance is due to Ucp1-mediated thermogenesis, finding no evidence for this mechanism.

4 citations

mousemetabolomics

𝕏 PostBluesky