Miriam B. Goodman

This study shows that the ECM gene mec-9 affects sensory cilia function and EV shedding in C. elegans through non-autonomous mechanisms.

This study shows valproic acid attracts C. elegans and activates chemosensory neurons via a cGMP signaling pathway involving tax-4, rGCs, and GPCRs.

This study shows that satiety, TAX-4, and OSM-9 channels regulate C. elegans attraction to microbial fermentation products and their biosynthetic precursors.

The paper introduces a platform to classify chemical cues by their chemosensory valence in C. elegans, identifying 37 plant small molecules that attract or repel wild-type worms.

This study measures touch-induced mechanical strain in C. elegans neurons and finds no effect of ECM mutations on strain transmission.

The paper shows that stronger touch stimuli recruit more distant mechano-electrical transduction channels in C. elegans, increasing touch response amplitude and kinetics.

This study develops a quantitative model to explain touch sensation in C. elegans by integrating tissue mechanics and neural responses to indentation stimuli.

The paper shows that CMK-1 in ASE neurons is essential for salt aversive learning in C. elegans by regulating calcium responses to salt.

The paper shows ultrasound induces behavioral responses in C. elegans through mechanical effects on neurons and MEC-4 ion channels, not thermal effects.